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In an interacting feed-back loop, CLOCK-BMAL1 complexes activate expression of nuclear receptors, REV-ERB α and RORα. Their protein items feedback to control BMAL1 by competitively binding retinoic acid-associated orphan receptor response factors from the BMAL1 promoter40. REV-ERBs repress the transcription of BMAL1, whereas RORs activate it. The
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